Severe Community-acquired Pneumonia Due to Staphylococcus aureus, 2003–04 Influenza Season
Jeffrey C. Hageman*
, Timothy M. Uyeki*, John S. Francis†, Daniel B. Jernigan*, J. Gary Wheeler‡, Carolyn B. Bridges*, Stephen J. Barenkamp§, Dawn M. Sievert¶, Arjun Srinivasan*, Meg C. Doherty†, Linda K. McDougal*, George E. Killgore*, Uri A. Lopatin#, Rebecca Coffman**, J. Kathryn MacDonald††, Sigrid K. McAllister*, Gregory E. Fosheim*, Jean B. Patel*, and L. Clifford McDonald*
Author affiliations: *Centers for Disease Control and Prevention, Atlanta, Georgia, USA; †Johns Hopkins Medical Institutions, Baltimore, Maryland, USA; ‡University of Arkansas for Medical Sciences College of Medicine, Little Rock, Arkansas, USA; §Saint Louis University School of Medicine, Saint Louis, Missouri, USA; ¶Michigan Department of Community Health, Lansing, Michigan, USA; #National Institutes of Health, Bethesda, Maryland, USA; **Oklahoma State Department of Health, Oklahoma City, Oklahoma, USA; ††Washington State Department of Health, Shoreline, Washington, USA
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Figure
Figure. Dendrogram of Staphylococcus aureus isolates determined by using SmaI–digested DNA recovered from patients with community-acquired pneumonia associated with influenzalike illness, influenza season, 2003–04. NA, not applicable (methicillin-susceptible); SE, staphylococcal enterotoxin A, B, C, H; REF, reference strain; PVL, Panton-Valentine leukocidin; TSST, toxin shock syndrome toxin; CHL, chloramphenicol; CLI, clindamycin; ERY, erythromycin; GM, gentamicin; LEV, levofloxacin; OX, oxacillin; PEN, penicillin; TET, tetracycline. *Inducible clindamycin resistance.
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